Horse Boost Ingredients: The Nine Names in the Blend

An Amazonian shrub, known in the trade as potency wood, and the first name inside the blend. The published record is thin and entirely preclinical: the most relevant paper is a cell study in which muira puama, singly and with three other agents, modulated the nitric oxide pathway in rat penile smooth muscle cells. That is a mechanism in a dish, not a result in a person.

No modern controlled human trial establishes a dose, which is why no dose comparison appears in the table below for this name. Its presence on the label is a traditional-use argument and is described that way here.

The best-studied name on this panel, and the one with the most useful result attached. A twelve-week randomised trial gave men aged 21 to 56 either 1,500 mg or 3,000 mg of gelatinised maca a day. Self-reported sexual desire rose from week eight; serum testosterone and oestradiol did not move, and the effect held after controlling for depression and anxiety scores.

That last part is the interesting half. Maca is routinely sold as a testosterone product and its own best trial reports the opposite. The systematic review of four randomised trials calls the overall evidence limited, and a 2024 review points out that black, red and yellow maca differ in composition, so the word on a label does not fix what is inside it. The chemistry survey sets out why.

A Brazilian bark with a long traditional reputation and a modern research record that stops at animals. The clearest paper is an antioxidant and antifatigue study in rodents; there is also in vitro work on central nervous system targets showing activity at monoamine transporters.

No human dose is established. Two different plants are sold as catuaba in South America, which is a known identity problem in this trade and another reason a name on a label is not a specification.

A catechin-bearing extract, and the one name here with a genuine safety literature rather than an efficacy one. The systematic review of green tea safety sets a safe intake of 338 mg of EGCG a day for a solid bolus dose and identifies the liver as the target organ at higher ones. Green tea also appears among the commoner botanical causes in the study of supplement-related liver injury.

None of that is an argument against 82 mg shared nine ways, which is a small fraction of those figures. It is an argument for adding it to any separate green tea extract you already take rather than counting them apart. On the benefit side, the review of green tea and human brain function finds effects mostly at doses well above anything a gummy carries.

The one amount this panel prints, and the reason it is worth printing is that green tea brings caffeine with it. Five milligrams is roughly a twentieth of a mug of brewed coffee.

The meta-analysis of thirty-one caffeine trials found small effects on accuracy and reaction time overall, with the clear separation at 200 mg and above. At 5 mg the honest statement is that nothing in the literature predicts an effect, and the figure is on the label so that anyone counting their intake can count it.

An adaptogen with the largest modern trial base on this panel, almost all of it about stress rather than vitality. The meta-analysis of twelve randomised trials in 1,002 people found reductions in anxiety and stress, put the favourable dose-response for stress at 300 to 600 mg a day, and rated the certainty of its own evidence as low. A separate meta-analysis of five sleep trials found the effect stronger at 600 mg a day and above, over eight weeks or more, and the cortisol review covers nine studies running 30 to 112 days.

Three hundred milligrams is the smallest of those figures and the whole blend here is 82 mg. NCCIH's page is the plain-language summary, and rare liver injury is catalogued in LiverTox.

An amino acid the body uses as a precursor for nitric oxide, which widens blood vessels. The meta-analysis of ten randomised trials in 540 men found improvement in mild to moderate erectile dysfunction at doses from 1,500 to 5,000 mg a day. A broader network meta-analysis of nutraceuticals places arginine among the better-supported options in a field where the bar is low.

Fifteen hundred milligrams is the bottom of that range. It is more than eighteen times the entire blend on this label, so nothing on this website suggests that result travels to this gummy.

On the bottle and absent from the seller's listing art. It is also the name with the most negative evidence attached, which is worth stating plainly. A 2025 review of ten studies in 483 men found doses of 400 to 750 mg a day improved erectile dysfunction in three of the five studies that measured it, while eight of the ten reported no significant change in androgen profile, and rated half the studies low quality. The earlier review titled Fact or Fiction reached the same conclusion about the androgen claim, and the systematic review of testosterone boosters covers the category the claim belongs to.

So tribulus is not a testosterone ingredient. It is on this label as part of a traditional combination, and this website makes no testosterone claim for it or for anything else.

The ninth name, also absent from the listing art. Its active flavonoid, icariin, is a weak phosphodiesterase-5 inhibitor in vitro, which is the same enzyme family prescription medicines act on. The clearest publication is an isolation of PDE5A-inhibiting flavonoids from Epimedium sagittatum, which is a test-tube result.

There is no modern human trial record worth quoting for this use, and the honest position is to say so rather than to dress up a weaker source. NCCIH publishes no page for it. The wider meta-analysis of herbal supplements sold for erectile dysfunction is the best available context.